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Induction of cell death by stimulation of protein kinase C in human epithelial cells expressing a mutant ras oncogene: a potential therapeutic target.
Hall-Jackson CA, Jones T, Eccles NG, Dawson TP, Bond JA, Gescher A, Wynford-Thomas D. Hall-Jackson CA, et al. Br J Cancer. 1998 Sep;78(5):641-51. doi: 10.1038/bjc.1998.554. Br J Cancer. 1998. PMID: 9744505 Free PMC article.
The cytotoxic effect of PMA could be blocked by bisindolylmaleimide (GF 109203X), a well-characterized inhibitor of c and n protein kinase C (PKC) isoforms, and by prior down-regulation of PKC, indicating that it is mediated by acute stimulation, rather than down-re …
The cytotoxic effect of PMA could be blocked by bisindolylmaleimide (GF 109203X), a well-characterized inhibitor of c and n protein k …
Effect of SB 203580 on the activity of c-Raf in vitro and in vivo.
Hall-Jackson CA, Goedert M, Hedge P, Cohen P. Hall-Jackson CA, et al. Oncogene. 1999 Mar 25;18(12):2047-54. doi: 10.1038/sj.onc.1202603. Oncogene. 1999. PMID: 10321729
One of the reasons for this is that SB 203580 also triggers a remarkable activation of c-Raf in vivo (when measured in the absence of the drug). The SB 203580-induced activation of c-Raf occurs without any increase in the GTP-loading of Ras, is not prevented by inhi …
One of the reasons for this is that SB 203580 also triggers a remarkable activation of c-Raf in vivo (when measured in the absence of …
Paradoxical activation of Raf by a novel Raf inhibitor.
Hall-Jackson CA, Eyers PA, Cohen P, Goedert M, Boyle FT, Hewitt N, Plant H, Hedge P. Hall-Jackson CA, et al. Chem Biol. 1999 Aug;6(8):559-68. doi: 10.1016/s1074-5521(99)80088-x. Chem Biol. 1999. PMID: 10421767
Paradoxically, exposure of cells to ZM 336372 induces > 100-fold activation of c-Raf (measured in the absence of compound), but without triggering any activation of MKK1 or p42 MAPK/ERK2. The ZM 336372-induced activation of c-Raf occurs without any increase in th …
Paradoxically, exposure of cells to ZM 336372 induces > 100-fold activation of c-Raf (measured in the absence of compound), but wi …
Multicenter automatic defibrillator implantation trial: reduce inappropriate therapy (MADIT-RIT): background, rationale, and clinical protocol.
Schuger C, Daubert JP, Brown MW, Cannom D, Estes NA 3rd, Hall WJ, Kayser T, Klein H, Olshansky B, Power KA, Wilber D, Zareba W, Moss AJ. Schuger C, et al. Ann Noninvasive Electrocardiol. 2012 Jul;17(3):176-85. doi: 10.1111/j.1542-474X.2012.00531.x. Ann Noninvasive Electrocardiol. 2012. PMID: 22816536 Free PMC article. Clinical Trial.
The objective of the MADIT-RIT trial is to determine if dual-chamber ICD or CRT-D devices with high rate cutoff (MADIT-RIT-Arm B) and/or long delay in combination with detection enhancements (MADIT-RIT-Arm C) are associated with fewer patients experiencing inappropriate th …
The objective of the MADIT-RIT trial is to determine if dual-chamber ICD or CRT-D devices with high rate cutoff (MADIT-RIT-Arm B) and/or lon …
SB203580 promotes EGF-stimulated early morphological differentiation in PC12 cell through activating ERK pathway.
New L, Li Y, Ge B, Zhong H, Mansbridge J, Liu K, Han J. New L, et al. J Cell Biochem. 2001;83(4):585-96. doi: 10.1002/jcb.1253. J Cell Biochem. 2001. PMID: 11746502
The pro-differentiation effect of SB203580 in EGF-treated PC12 cells was found to be independent of its function of p38 inhibition but was through an effect on the ERK pathway that has been recently reported (Kalmes et al. [1999] FEBS Lett. 444: 71-74; Hall-Jackson …
The pro-differentiation effect of SB203580 in EGF-treated PC12 cells was found to be independent of its function of p38 inhibition but was t …
Location of mutation in the KCNQ1 and phenotypic presentation of long QT syndrome.
Zareba W, Moss AJ, Sheu G, Kaufman ES, Priori S, Vincent GM, Towbin JA, Benhorin J, Schwartz PJ, Napolitano C, Hall WJ, Keating MT, Qi M, Robinson J, Andrews ML; International LQTS Registry, University of Rochester, Rochester, New York. Zareba W, et al. J Cardiovasc Electrophysiol. 2003 Nov;14(11):1149-53. doi: 10.1046/j.1540-8167.2003.03177.x. J Cardiovasc Electrophysiol. 2003. PMID: 14678125
Demographic, clinical, and follow-up information was compared among subjects with different locations of KCNQ1 mutations defined as pre-pore region including N-terminus (1-278), pore region (279-354), and post-pore region including C-terminus (>354). Cardiac events obse …
Demographic, clinical, and follow-up information was compared among subjects with different locations of KCNQ1 mutations defined as pre-pore …
Arsenite blocks growth factor induced activation of the MAP kinase cascade, upstream of Ras and downstream of Grb2-Sos.
Doza YN, Hall-Jackson CA, Cohen P. Doza YN, et al. Oncogene. 1998 Jul 9;17(1):19-24. doi: 10.1038/sj.onc.1202168. Oncogene. 1998. PMID: 9671310
Pretreatment of cells with 0.5 mM sodium arsenite (but not other activators of stress-activated MAP kinase cascades) prevents the activation of p21Ras and strongly suppresses the activation of c-Raf and the MAP kinase cascade by a variety of growth factors. ...
Pretreatment of cells with 0.5 mM sodium arsenite (but not other activators of stress-activated MAP kinase cascades) prevents the activation …